Thursday, December 26, 2019

How to Scan and Mark Latin Poetry

To learn to scan a line of Latin poetry, it helps to know the meter and to use a text that shows the macrons. Lets assume you have a text of the beginning of The Aeneid with macrons. Since it is an ancient epic, The Aeneid is in dactylic hexameters, which is a meter the AP exams typically expect you to know. Find the Long Syllables First, you mark all syllables that are long by nature. Syllables that are long by nature are those with diphthongs, ae, au, ei, eu, oe, and ui. Those syllables with macrons over the vowels are long by nature. For simplicity, a circumflex will be used for a macron here. (Macrons are usually long marks †¾ over the vowels, but you use the long mark †¾ over the syllables vowel to mark the syllable as long when you scan your lines.) Tip: For an AP exam, the help offered by the macron will probably not be available, so when you use a Latin dictionary to look up a word, make note of the long vowels. 3 Consecutive Vowels If there are 3 vowels in a row:and there is a macron over one of the vowels, it is not part of the diphthong; thus, dià ªÃƒ ®, which has two macrons, has no diphthongs. Dià ªÃƒ ® has 3 syllables: di, à ª, and à ®.and the second and third vowels form a diphthong, the preceding vowel is short. (This 1st vowel is also short if there are 2 vowels that do not form a diphthong.)Next, find and mark as long all the syllables that are long by position. Double Consonants Those syllables in which the vowel is followed by two consonants (one or both of which may be in the next syllable) are long by position.A syllable that ends in X or (sometimes) Z is long by position because X or (sometimes) Z counts as a double consonant. Extra Linguistic Information: The 2 consonant sounds are [k] and [s] for X and [d] and [z] for Z.However, ch, ph, and th do not count as double consonants. They are the equivalent of the Greek letters Chi, Phi, and Theta.For qu and sometimes gu, the u is really a glide [w] sound rather than a vowel, but it doesnt make the q or g into a double consonant.When the second consonant is an l or an r, the syllable may or may not be long by position. When the l or r is the first consonant, it counts towards the position. Extra Linguistic Information: The consonants [l] and [r] are called liquids and are more sonorant (closer to vowels) than stop consonants [p] [t] and [k]. Glides are even more sonorant.When a word ends in a vowel or a vowe l followed by an m and the first letter of the next word is a vowel or the letter h, the syllable ending in a vowel or an m elides with the next syllable, so you dont mark it separately. You may put a line through it.Extra Linguistic Information: The [h] counts as aspiration or rough breathing in Greek, rather than a consonant. Scan a Line of Latin Lets look at an actual line of Latin: Arma virumque canà ´, Trà ´iae quà ® prà ®mus ab à ´rà ®s Can you find the 7 syllables that are long by nature? There are 6 macrons and 1 diphthong. Mark them all as long. Here they are bolded; syllables are separated from each other: Ar-ma vi-rum-que ca- nà ´, Trà ´-iae quà ® prà ®-mus ab à ´-rà ®s Notice that in Trà ´iae there is a diphthong, a macron, and an i in between. More Information: This intervocalic i acts as a consonant (j), rather than a vowel. How Many Syllables Are Long by Position? There are only 2: Ar-maThe two consonants are r and m.vi-rum-quethe two consonants are m and q. Here is the line with all the long syllables noted: Ar-ma vi-rum-que ca-nà ´, Trà ´-iae quà ® prà ®-mus ab à ´-rà ®s Mark According to the Known Meter Since you already know this is an epic and in the meter called dactylic hexameter, you know you should have 6 feet (hexa-) of dactyls. Dactyl is a long syllable followed by two shorts, which is exactly what you have at the start of the line: Ar-ma vi-You may put short marks over the 2 short syllables. (If you arent bolding the long syllables, you should mark the shorts, perhaps with a Ï…, and mark the longs with a long mark †¾ over them: †¾Ãâ€¦Ãâ€¦.) This is the first foot. You should put a line (|) after it to mark the foots end.The next and all succeeding feet begin with a long syllable as well. It looks as though the second foot is as simple as the first:rum-que ca-The second foot is just like the first. No problem so far, but then look what comes next. Its all long syllables:nà ´, Trà ´-iae quà ® prà ®Have no fear. There is an easy solution here. One long syllable is the equivalent of 2 shorts. (Mind you, you cant use two shorts for the start of a dactyl.) Therefore, a dactyl can be long, short, short, or long, long and thats what weve got. The long, long syllable is called a spondee, so technically, you should say that a spondee can substitute for a dactyl.nà ´, Trà ´iae quà ® and then prà ® b ecomes the long syllable in a regular dactyl:prà ®-mus ab We just need one more syllable to make the 6 dactyls of a line of dactylic hexameter. What we have left is the same pattern we saw for the 3rd and 4th feet, two longs:à ´-rà ®sOne extra bonus is that it doesnt matter whether the final syllable is long or short. The final syllable is an anceps. You can mark the anceps with an x.Tip: This customary †¾ x final foot makes it possible to work backward from the last two syllables  if the passage is tricky. You have now scanned a line of dactylic hexameter: Ar-ma vi-|rum-que ca-|nà ´, Trà ´-|iae quà ® |prà ®-mus ab| à ´-rà ®s†¾Ãâ€¦Ãâ€¦ | †¾Ãâ€¦Ãâ€¦ | †¾ †¾ | †¾ †¾ |†¾Ãâ€¦Ãâ€¦ |†¾x Line With Elision The third line of the first book of The Aeneid offers examples of elision twice in succession. If you are speaking the lines, you dont pronounce the italicized elided parts. Here, the syllable with the ictus is marked with an acute accent and the long syllables are bolded, as above: là ­-to-ra | mà ºl- tum il-| le à ©t ter-| rà ­s jac-| tà ¡-tus et| à ¡l- to†¾Ãâ€¦Ãâ€¦ | †¾ †¾ | †¾ †¾ | †¾ †¾ |†¾Ãâ€¦Ãâ€¦ |†¾xSyllables Read: li-to-ra-mul-til-let-ter-ris-jac-ta-tus-et-al-to References: Guide to Scansion of Latin PoetryGildersleeves Latin Grammar

Wednesday, December 18, 2019

An Analysis On Financial Struggles - 1122 Words

College Admissions: An Analysis on Financial Struggles As inflation in the United States continues to rise, as does the cost of many things, specifically college fees. In a world such as the one we live in today, it is extremely hard to attain a successful career without a college degree. Although, one may end up spending their entire life trying to pay off their college debts in order to get a degree. While the knowledge and skills attained during college are extremely beneficial later on in life, many students are unable to go to college due to financial issues. The prices have skyrocketed and left many graduates in debt for years later. Many high school students who plan to attend college in the future, often work an after school job in order to get the money they will be needing for college. College is so expensive due to the tuition as well as room and board with all the necessities and one way I have made financial plans for college is by working an after school job. Tuition for colleges varies between every state and school, but m ost are quite expensive. Especially, if one plans to attend college out of the state they are residing in. It has been calculated that the average annual cost of study in the United States is between $20,000 and $35,000. Evidently, college tuition is already high, but the level and location of the college can also increase this expense. Tution itself is already pricey, but there are also many fees that must be included. For example, evenShow MoreRelatedThe Return Of Depression Economics1437 Words   |  6 Pagesroots of modern and prior financial crisis economics. In his book, The Return of Depression Economics and The Crisis of 2008, Krugman first educates the reader of historical and foreign financial crises which allows for a deeper understanding of the modern financial system. 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Tuesday, December 10, 2019

Effects Of The Shock On Different Systems â€Myassignmenrthelp.Com

Question: Discuss About The Effects Of The Shock On Different Systems? Answer: Introduction Patient Maureen, 77 years old an Australian woman. Presents with what seems to be in the progressive phase of hypovolemic shock. The symptoms she presents are as a result of the effects of the shock on the different systems. The respiratory, nervous, cardiovascular, renal and hepatic systems. The breathing is at 28 breaths per minute. This breath must be shallow as the saturation level of oxygen is undetectable. The heart rate is at 120 beats a minute. Despite this high beats the systolic blood pressure is at 80mmHg. There is evidence of vasoconstriction on the skin as the capillary fill lasts more than 4seconds. This is a classical evidence of shunting as the blood flow is redirected to other vital organs. She presents with hypovolemia. The hemoglobin levels are below average. The blood cells are also below the average. She has signs of ascites and vomits blood which is a huge contributor of fluid volume deficiency. The vomiting is as a result of ulcers on the gastrointestinal as a result of the vasoconstriction. She is also on diclofenac which worsens the ulcers because of its pharmacodynamics. She is pale and unconscious all this points to hypovolemic shock. Physiology of normal fluid balance/distribution. Brunner and Saddarth (2013), states that around 60% of an adults body weight is composed of fluids, that is, water and electrolytes. This amount is not fixed. It ranges from one person to the other. Gender, age, and body fats are some of the factors that determine the amount of body fluid. Lewis et al (2014), from the book it has been researched and proven that men have more body fluids that women, Young people has more fluids when compared with old people and the obese people have less water. (Sloan 2013), According to the book, the body fluids are located in two compartments. The intracellular fluid, that is, the fluid in the cells and the extracellular space. Barret et al (2017), further classifies this compartment. According to them, the intracellular holds two-thirds of the fluids while the extracellular holds the remaining third. They go on explaining that the extracellular fluid is further divided into interstitial, intravascular and trans cellular fluids. The fluid volume is maintained by the body. The body is equipped with mechanisms to help it balance the fluids. The homeostasis mechanisms. These include the kidney function. The heart and blood vessels function, lung functions, pituitary function, renin angiotensin aldosterone system, parathyroid function, Anti-diuretic hormone and thirst, baroreceptors and the osmoreceptors. These mechanisms ensure that there is a fluid balance because if this balance gets altered it can cause a lot of harm. Hypovolemia Brunner and Saddarth (2013), explains that hypovolemia or fluid volume deficit takes place when extracellular fluid volume loss exceeds the volume of the fluid intake. The water and the electrolytes are lost in the same proportion making a deficit in both the extracellular and the intracellular. Marcin (2016), adds that there is a difference between dehydration and hypovolemia. In dehydration, water is the only thing lost. Fluid volume deficit is accelerated with decreased intake of water. Causes of fluid volume deficit other than inadequate water intake include; vomiting, sweating, diarrhea, gastrointestinal suctioning, Beck, (2015). If the homeostasis mechanisms, for example, the kidney functions are impaired due to a disease, this could cause the fluid volume deficit. A patient with fluid volume deficit has the following presentations: they have an acute weight loss, their skin turgor is decreased, they have oliguria, this small amount of urine is highly concentrated, they present with hypovolemia, their heart rates are fast but weak, the neck veins are flattened, they have increased temperature, their central venous pressure is decreased, they are cool, they have clammy skin due to the vasoconstriction of the vessels peripherally, they are thirsty have muscle cramps and weakness. If this condition is not reversed it can worsen and cause shock which in turn causes death. Shock Mikhail (2015), Defines shock as a condition that is life threatening. It is usually as a result of inadequate perfusion of tissues. If untreated it causes death. The systemic blood pressure is unable/inadequate in delivering the required amount of oxygen and nutrients to enhance cellular and vital organ function, Balk (2015), Inadequate perfusion ( inadequate oxygen and nutrients), this causes cellular starvation, this progresses to cell death, in turn it causes organ dysfunction which progresses to organ failure and later on death. As mentioned shock is fatal. It progresses rapidly or slowly and it affects all body systems. Hypovolemic shock Jindal et al., (2014), hypovolemic shock is the most common which results in a decrease of 15%-25% of intravascular volume. As mentioned earlier intravascular fluid represents the blood which is an extracellular fluid. The percentage will present a loss of 750 ml to 1300ml of blood given that in a normal physiological man weighing 70kgs has 5lts to 6lt of blood. Hypovolemic shock can be caused by external fluid losses. This could be a traumatic loss of blood (internal bleeding, a wound, GIT bleeding) or internal fluid shift which could be as a result of severe edema, severe dehydration, ascites. Rauen and Munron, (2016), they vividly explain how hypovolemic shock occurs. It starts when there is a deficit in the intravascular fluid. This deficit causes a decrease in the venous return. Due to this reduction, there is a reduced ventricular refilling which in turns causes a reduction in stroke volume and cardiac output. When this happens the blood pressure drops. This compromises the tis sue perfusion which progresses to death. The manifestation of the patient depends on which stage they are in. Stages of hypovolemic shock as per the physical assessment (ABCDE) Through the assessment of airway, breathing, circulation, disability and exposure, there are three stages of shock. All systems are assessed. The cardiovascular; that is the systolic blood pressure and the heart rate. The respiratory system checks the breathing rate. The skin for warmth and liver function. The kidney functions. All this are checked up and the findings helps categorize the patient. The first stage is compensatory. The body tries to normalize the fluids. At this phase, the body displays the normal fight or flight response. The heart rate increases, vasoconstriction occurs, the hearts contractility increases, there is a shunt of blood flow to other organs. Blood is redirected to the vital organs, that is, the brain, liver, and kidney. This makes the patients skin feel cold and clammy. The patients blood pressure is normal at this stage, heart rate is above 100 beats per minute, the respiratory rates is above 20breaths per minute, there is reduced urinary output, the pat ient is confused, laboratory findings reveal respiratory alkalosis. At this stage, the body does its best to have a homeostasis. If it is not restored the shock progresses to the next phase which is the progressive stage. Abraham et al., (2015), at this stage the body systems are unable to maintain the blood pressure. The blood pressure falls beyond the average range of the systolic blood pressure. It falls beyond 90 mm Hg. The manifestation of shock at this stage is usually due to two things. One the overworking heart becomes dysfunctional hence poor/ no perfusion to the muscles. This brings about ischemia. Two, the microcirculation auto regulatory function fails because of the biochemical mediators. This makes the venous and arterioles to constrict making the perfusion poorer, Kumar and Haery (2014). The systolic blood pressure is below 80mmHg. The heart rate is above 150beats per minute. The breathing is shallow, rapid and with crackles. The skin has petechial and is mottled. The urinary output is 0.5ml/kg/hr. The patient is lethargic. Lab findings reveal metabolic acidosis. This is the stage where patient Maureen is at. Airway is being compromised by the nervous system as she is semiconscious. Breathing rate is very high although the breaths are shallow. Circulation is not normal as the capillary refill time has lengthen, skin is pale and cold and systolic pressure is at 80mmHg. Disability the patient is at risk of death as circulation and breathing has been compromised. Exposure, her organ are exposed to death if this stage is not reversed. If this stage is not reversed the patient progresses to irreversible phase. At this stage, the cell damage is so severe. The patient is not responsive to any treatment. The patient cannot survive. The patient presents with jaundice due to liver failure. They require mechanical or pharmacological support for the heart function. They require intubation for breathing. They have anuria and requires dialysis. They are uncon scious and have a profound acidosis. The heart rate is erratic or asystole. Patient Maureen Interventions using ISBAR ISBAR stands for: identification, situation, background, assessment and recommendation. I, Mary, a registered nurse, rotating at the casualty and emergency ward, have assessed patient Maureen. She appears to be in progressive stage of hypovolemic shock. The following are identifies as to why she is in shock. One, her airway/breathing is compromised. She is semiconscious so she requires assistance in breathing. Her breathing rate is so high and her breaths are shallow. Her systolic blood pressure is at 80mmHg. It is hypovolemic shock because she has signs of low hemoglobin count. From the lab investigations her hemoglobin was at 9g/l. She lost blood from vomiting. Has signs of ascites which causes fluid imbalance. The interventions or recommendations that are most appropriate for patient Maureen will be targeting the following. One is to optimize the intravascular volume. The second target is to support the pumping function of the heart. The third thing is to improve the competence an d functioning of the vascular system. Fourth is to redistribute the fluid volume and lastly is to find a solution to what is causing the fluid volume deficit. If it is vomiting, anti-emetics will be administered. If it is fluid intake deficiency, fluid replacement therapy is done. Choi et al, (2015), for shock management fluid replacement is necessary so as to restore intravascular volume. This includes fluids like 0.9% sodium chloride and lactated Ringers solution. Fein and Calalang (2015), vasoactive medications are important in restoring the vasomotor tone and improving the functioning of the heart. This drug includes, sympathomimetic, vasodilators and vasoconstrictors. Nutritional support is required to take care of metabolic deficiency. During the compensation phase, there was a release of catecholamine's which causes depletion of the stored glycogen. Administration of nutrients is administered as soon as possible. Anti-acids and histamine blockers are administered to help with the stress ulcers. Conclusion From the above, it is clear how hypovolemia develops. How it progresses to hypovolemic shock. The stages of the hypovolemic shock and the manifestations at each stage clarifying why they occur. The organ systems effects during shock. The role of a nurse during shock. With all that knowledge it was easy to relate it to the case study that was presented. Maureen is at progressive shock stage. She has all the signs and symptoms. The shock could have resulted from the effects of the medications she is on. They could have caused stomach ulcers which caused blood loss. The other possible cause is the ascites. The interventions were mentioned and if followed to the later the patient might be able to recover. References Abraham, E., Matthay, M. A., Dinarello, C. A., et al. (2014). Consensus conference definitions for hypovolemia, hypovolemic shock, acute lung injury, and acute respiratory distress syndrome: Time for a reevaluation. Critical Care Medicine, 28 (1), 232235 Balk, R. A. (2015). Pathogenesis and management of multiple organ dysfunctions or failure in severe hypovolemia and hypovolemic shock. Critical Care Clinics, 16(2), 337351. Barrett, E., Barman, M., Boitano, S., (2017) Ganongs review of medical physiology. Lange basic science. 24th edition. Beck, L. H. (2015). The aging kidney. Defending a delicate balance of fluid and electrolytes. Geriatrics, 55(4), 2628, 3132. Brunner and Suddarth, (2013). Textbook of medical and surgical nursing, 13th edition Choi, P. T., Yip, G., Quinonez, L. G., Cook, D. J. (2014). Crystalloids vs. colloids in fluid resuscitation: A systematic review. Critical Care Medicine, 27(1), 200209. Fein, A. M., Calalang-Colucci, M. G. (2014). Acute lung injury and acute respiratory distress syndrome in sepsis and septic shock. Critical Care Clinics, 16(2), 289313. Guyton, A. C. (2015). Textbook of medical physiology (13th ed.). Philadelphia: W. B. Saunders Jindal, N., Hollenberg, S. M., Dellinger, R. P. (2015). Pharmacologic issues in the management of hypovolemic shock. Critical Care Clinics, 16(2), 233248. Kreimeier, U. (2016). Pathophysiology of fluid imbalance. Critical Care (London), 8, Suppl 2:S3S7. Kumar, A., Haery, C., Parrillo, J. E. (2015). Myocardial dysfunction in hypovolemic shock. Critical Care Clinics, 16(2), 251281 Lewis, l., Dirksen, R., McLean, M., (2013) medical-surgical nursing: assessment and management of clinical problems, 8th edition. Marcin, J., (2016). Hypovolemic shock. Medical review journal. 21st edition. McKinley, M. G. (2014). Shock, Introduction to critical care nursing (10th ed.). Philadelphia: W. B. Saunders. Mikhail, J. (2015). Resuscitation endpoints in trauma. AACN Clinical Issues, 32(1), 1021. Rauen, C. A., Munro, N. (2015). Shock. In M. R. Kinney, S. B. Dunbar, J. A. Brooks-Brunn, N. Moleter, J. M. Vitello-Cicciu (Eds.), (2013). AACNs clinical reference for critical care nursing (12th ed.). St. Louis: Mosby Vincent, J. L., Ferreira, F. L. (2016). Evaluation of organ failure: We are making progress. Intensive Care Medicine, 26(6), 1023 1024

Monday, December 2, 2019

Value and Fast Food Customers free essay sample

What situation did Skinner inherit when he became CEO? What are the current forces in the external environment that affect Skinner’s ongoing strategy? 2. What source of competitive advantage does McDonald’s have, and is that position supported by its value chain and other internal resources? -Inherit the previous CEO Cantalupo’s turnaround strategy. This strategy referred as the †Plan to win† tried to target various critical areas that needed to be addressed. -Rapid market fragmentation, which is describing the changes of consumer taste have made once-exotic foods like sushi and burritos everyday options. Many fast food customers are looking for healthier and better tasting food. Moreover, competitions has been coming from quick meals of all sorts that can be found in supermarkets, convenience stores and vending machines. Demographic customers now working around theclock, expecting 24 hour access  to fast food, how toplease range of customers from kids to  contractors? Sociocultural customers preferences have changed tomore exotic foods, healthier food with better taste Economic current economic downturn means customers might be trading down  to McDonald’s if  they ant to eat out Global boundaries are disappearing, travelers moreopen to global consistency in food offerings GoldenArches are accepted, and expected, everywhere 2. Cost leadership has been the traditional strategy for thefast- food industry, but McDonald’s kept costs under control in order to achieve parity with competitors -McDonald’s tried to develop a differentiationadvantagewhile keeping costs at a reasonable level -Differentiation requires the creation of something that isperceived industry-wide as unique and valued bycustomers -Differentiation s achieved by a firm configuring  its valuechain activities to support its position so customers arewilling to pay a premium for something unique – could McDonald’s do this effectively? Value-Chain  Analysis: -Sequential process of value-creating activities -The amount that buyers are willing  to pay forwhat a firm provides them -Value is measured by total revenue -Firm is profitable to  the extent the value itreceives exceeds the total costs involved increating its product or service Value  ChainActivity How does McDonald’s create value? Primary: Inbo und  logistics: Hard  to  assess Operations:   Strived  for  consistency  across  the  chain,  withdiffering results. Refurbishing of restaurants,change in hours may help draw customers. Outbound  logistics:   Hard  to  assess Marketing and sales: Many product innovations failed, $1 menu  didn’t go well with franchisees. I’m Loving It campaign was attempt to reach all customers. Service:   Hard  to  assess Value  ChainActivity How does McDonald’s create value? Secondary: Procurement:   Info  not  available  in  the  case Technology development: Adoption of expensive cooking processesfailed to generate desired results.